![]() VZV myelitis often develops within the 3 weeks following the appearance of a skin rash. VZV can cause radicular pain without skin lesions (zoster sine herpete, ZSH), sensorimotor neuropathy, and facial nerve paralysis. While the prevalence of these complications among healthy individuals ranges from 0.1% to 0.3%, it increases to 35% in immunocompromised patients. Neurological complications of herpes zoster include vasculitis-induced cerebral infarction, meningoencephalitis, segmental sensory disturbance, facial-nerve palsy (Hunt syndrome), myelitis, and postherpetic neuralgia. Its primary infection causes chickenpox, after which the virus establishes a lifelong latency in cranial nerves and the dorsal root ganglia and can reactivate as herpes zoster. The varicella-zoster virus (VZV) is a DNA virus of the herpesvirus family.
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